A case report of intracortical fibrous dysplasia.
نویسندگان
چکیده
منابع مشابه
Monostotic Fibrous Dysplasia: A Case Report
Fibrous dysplasia (FD) is a benign fibro-osseous bone disease of unknown etiology and uncertain pathogenesis. When bone maturation is completed, indicating the occurence of stabilization is a strong evidence of mechanism. The lesion frequently affects the craniofacial skeleton. The maxilla is affected twice comparing mandible and occurs more frequently in the posterior area. In this case, a 16 ...
متن کاملMonostotic fibrous dysplasia (a case report).
Fibrous dysplasia is a benign fibro-osseous lesion of the bone that commonly affects the jaws with a higher prevalence in the maxilla than the mandible. It is a lesion of unknown etiology, uncertain pathogenesis, and diverse histopathology. Fibrous dysplasia can involve multiple bones (polyostotic) or a single bone (monostotic). It represents about 2 to 5% of all bone tumors and over 7% of all ...
متن کامل[Craniofacial fibrous dysplasia: a case report].
Fibrous dysplasia is classified among benign bone tumors. It is a bone development anomaly characterized by hamartoma proliferation of fibrous tissue within the medullary bone, with secondary bony metaplasia, producing immature, newly formed and weakly calcified bone, without maturation of the osteoblast. It can be monostotic or polyostotic. The craniofacial localization occurs in 10%-25% of ca...
متن کاملPolyostotic fibrous dysplasia; a case report.
Fibrous dysplasia is a condition leading to abnormal differentiation of osteoblasts which leads to replacement of normal bone with fibrous stroma [1-3]. It may involve single bone (monostotic) or multiple bones (polyostotic). It may affect any bone however; skull and ribs are the commonest [2,4-6]. It is usually asymptomatic and is an incidental finding. It may become symptomatic when complicat...
متن کاملMalignant transformation of fibrous dysplasia: A case report
Secondary osteosarcoma from fibrous dysplasia (FD) is very rare. The etiology of FD is linked to activating missense mutations of the guanine nucleotide-binding protein α-subunit (GNAS) gene, which encodes the stimulatory α subunit of the G protein (Gsα) and is located at chromosome 20q13. These mutations are central to the pathogenesis of FD; however, it is not known whether Gsα mutations are ...
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ژورنال
عنوان ژورنال: Orthopedics & Traumatology
سال: 1984
ISSN: 1349-4333,0037-1033
DOI: 10.5035/nishiseisai.33.154